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The gene INFLORESCENCE DEFICIENT IN ABSCISSION
(IDA) gives a striking
floral abscission phenotype in the model plant
Arabidopsis thaliana when mutated. Prior to this
publication the only Arabidopsis mutants delayed in
floral organ abscission that had been characterized
in any detail were the ethylene insensitive mutants
etr1 and ein2 in addition to an antisense line of
the receptor-like kinase HAESA and an
overexpression line of the MADS-box AGL15. The
work presented
here suggest that although
ethylene has an important role in regulating the
timing of floral organ abscission it is not an
essential component of the pathway leading to floral
shedding. By performing in depth
studies of the mutant ida we wanted to determine
whether a gene that was an essential
component in the regulation of floral abscission was
involved in ethylene signaling or
perception, and if not what roles it played during
the abscission process.
(IDA) gives a striking
floral abscission phenotype in the model plant
Arabidopsis thaliana when mutated. Prior to this
publication the only Arabidopsis mutants delayed in
floral organ abscission that had been characterized
in any detail were the ethylene insensitive mutants
etr1 and ein2 in addition to an antisense line of
the receptor-like kinase HAESA and an
overexpression line of the MADS-box AGL15. The
work presented
here suggest that although
ethylene has an important role in regulating the
timing of floral organ abscission it is not an
essential component of the pathway leading to floral
shedding. By performing in depth
studies of the mutant ida we wanted to determine
whether a gene that was an essential
component in the regulation of floral abscission was
involved in ethylene signaling or
perception, and if not what roles it played during
the abscission process.