Management of Generalized Dermatitis in Clinical Practice translates the mechanisms of dermatitis from basic science evidence to practice based recommendations for clinical care. The role of allergic contact dermatitis in atopic dermatitis is explored in depth. Primary care physicians, allergists, and dermatologists will enjoy the fresh perspective that moves beyond treatment with corticosteroids and provides diagnostic and therapeutic algorithms for this complex condition.
Management of Generalized Dermatitis in Clinical Practice translates the mechanisms of dermatitis from basic science evidence to practice based recommendations for clinical care. The role of allergic contact dermatitis in atopic dermatitis is explored in depth. Primary care physicians, allergists, and dermatologists will enjoy the fresh perspective that moves beyond treatment with corticosteroids and provides diagnostic and therapeutic algorithms for this complex condition.
Susan Nedorost, Associate Professor in the Department of Dermatology and Director of the Environmental and Occupational Dermatology Clinic, University Hospitals Case Medical Center, Cleveland, OH, US.
Inhaltsangabe
Section I.Common examples.- 1.Auto-eczematization from stasis dermatitis.- 2.Systemic contact dermatitis.- 3.Protein contact dermatitis and food pollen syndromes.- 4.Atopic dermatitis as a model for protein contact dermatitis.- 5.Sensitization to food via initial exposure on inflamed perioral skin instead of. Section II: Less common examples.- 1.Erythema multiforme complicating contact dermatitis.- 2.Hypereosinophilic syndrome.- 3.Eczematous drug eruption.-Section III: Mimics in Differential Diagnosis.- 1.Bullous pemphigoid.- 2.Scabies.- 3.Widespread allergic contact dermatitis
Section I.Common examples.- 1.Auto-eczematization from stasis dermatitis.- 2.Systemic contact dermatitis.- 3.Protein contact dermatitis and food pollen syndromes.- 4.Atopic dermatitis as a model for protein contact dermatitis.- 5.Sensitization to food via initial exposure on inflamed perioral skin instead of. Section II: Less common examples.- 1.Erythema multiforme complicating contact dermatitis.- 2.Hypereosinophilic syndrome.- 3.Eczematous drug eruption.-Section III: Mimics in Differential Diagnosis.- 1.Bullous pemphigoid.- 2.Scabies.- 3.Widespread allergic contact dermatitis
Section I.Common examples.- 1.Auto-eczematization from stasis dermatitis.- 2.Systemic contact dermatitis.- 3.Protein contact dermatitis and food pollen syndromes.- 4.Atopic dermatitis as a model for protein contact dermatitis.- 5.Sensitization to food via initial exposure on inflamed perioral skin instead of. Section II: Less common examples.- 1.Erythema multiforme complicating contact dermatitis.- 2.Hypereosinophilic syndrome.- 3.Eczematous drug eruption.-Section III: Mimics in Differential Diagnosis.- 1.Bullous pemphigoid.- 2.Scabies.- 3.Widespread allergic contact dermatitis
Section I.Common examples.- 1.Auto-eczematization from stasis dermatitis.- 2.Systemic contact dermatitis.- 3.Protein contact dermatitis and food pollen syndromes.- 4.Atopic dermatitis as a model for protein contact dermatitis.- 5.Sensitization to food via initial exposure on inflamed perioral skin instead of. Section II: Less common examples.- 1.Erythema multiforme complicating contact dermatitis.- 2.Hypereosinophilic syndrome.- 3.Eczematous drug eruption.-Section III: Mimics in Differential Diagnosis.- 1.Bullous pemphigoid.- 2.Scabies.- 3.Widespread allergic contact dermatitis
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