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In children with cerebral ischemia hemostasis changes consisted in decreased hemotocrit and hemoglobin levels, increased levels of ACTV and RFMC compared with the control group. The group of premature infants with intracranial hemorrhages of hypoxic genesis of CP CNS revealed increased AChTV, TV, RFMC and FG against the background of decreased hematocrit, hemoglobin and PV, which excludes the local pathological process in hypoxic hemorrhagic lesions of CNS. In children with DIC all baseline tests changed, there was a consumption of coagulation factors, FG and ACTV, increased fibrinolysis compared to the group of children with intracranial hemorrhages of hypoxic genesis of CP CNSDDapathogenetic model of CP CNS development was developedby logistic regression to identify relative risk factors for the development of DIC. The use of this model in the dynamics of the early neonatal period in premature infants can help justify the individual duration of vasopressor and hemostatic therapy to prevent the development of severe CP CNS.