Leading researchers are specially invited to provide a complete understanding of the key topics in these archetypal multidisciplinary fields. In a form immediately useful to scientists, this periodical aims to filter, highlight and review the latest developments in these rapidly advancing fields.
Leading researchers are specially invited to provide a complete understanding of the key topics in these archetypal multidisciplinary fields. In a form immediately useful to scientists, this periodical aims to filter, highlight and review the latest developments in these rapidly advancing fields.
Produktdetails
Produktdetails
Reviews of Physiology, Biochemistry and Pharmacology 170
Intrauterine Growth Retardation (IUGR) as a novel condition of Insulin-like growth factor-1 (IGF-1) deficiency.- CFTR: A new horizon in the pathomechanism and treatment of pancreatitis.- Signature and pathophysiology of non-canonical pores in voltage-dependent cation channels.- Cardiac response to oxidative stress induced by mitochondrial dysfunction.
Intrauterine Growth Retardation (IUGR) as a novel condition of Insulin-like growth factor-1 (IGF-1) deficiency.- CFTR: A new horizon in the pathomechanism and treatment of pancreatitis.- Signature and pathophysiology of non-canonical pores in voltage-dependent cation channels.- Cardiac response to oxidative stress induced by mitochondrial dysfunction.
Intrauterine Growth Retardation (IUGR) as a novel condition of Insulin-like growth factor-1 (IGF-1) deficiency.- CFTR: A new horizon in the pathomechanism and treatment of pancreatitis.- Signature and pathophysiology of non-canonical pores in voltage-dependent cation channels.- Cardiac response to oxidative stress induced by mitochondrial dysfunction.
Intrauterine Growth Retardation (IUGR) as a novel condition of Insulin-like growth factor-1 (IGF-1) deficiency.- CFTR: A new horizon in the pathomechanism and treatment of pancreatitis.- Signature and pathophysiology of non-canonical pores in voltage-dependent cation channels.- Cardiac response to oxidative stress induced by mitochondrial dysfunction.
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