B. Brüne et al.: Protein Thiol Modification and Apoptotic Cell Death as cGMP-Independent Nitric Oxide. H. Esterbauer and P. Ramos: Chemistry and Pathophysiology of Oxidation of LDL. C. Götz and M. Montenarh: DNA Damage, DNA Repair, and Apoptosis. E.R. Werner et al.: Biosynthesis of Nitric Oxide: Dependence on Pteridine Metabolism. R. Bernhardt: Cytochrome P450: Structure, Function, and Generation of Reactive Oxygen Species. B. Epe. DNA Damage Profiles Induced by Oxidizing Agents
B. Brüne et al.: Protein Thiol Modification and Apoptotic Cell Death as cGMP-Independent Nitric Oxide. H. Esterbauer and P. Ramos: Chemistry and Pathophysiology of Oxidation of LDL. C. Götz and M. Montenarh: DNA Damage, DNA Repair, and Apoptosis. E.R. Werner et al.: Biosynthesis of Nitric Oxide: Dependence on Pteridine Metabolism. R. Bernhardt: Cytochrome P450: Structure, Function, and Generation of Reactive Oxygen Species. B. Epe. DNA Damage Profiles Induced by Oxidizing AgentsHinweis: Dieser Artikel kann nur an eine deutsche Lieferadresse ausgeliefert werden.
Produktdetails
Produktdetails
Reviews of Physiology, Biochemistry and Pharmacology 127
Protein thiol modification and apoptotic cell death as cGMP-independent nitric oxide (NO) signaling pathways.- Chemistry and pathophysiology of oxidation of LDL.- p53: DNA damage, DNA repair, and apoptosis.- Biosynthesis of nitric oxide: Dependence on pteridine metabolism.- Cytochrome P450: Structure, function, and generation of reactive oxygen species.- DNA damage profiles induced by oxidizing agents.- Depolarization - Transcription coupling in excitable cells.
Protein thiol modification and apoptotic cell death as cGMP-independent nitric oxide (NO) signaling pathways.- Chemistry and pathophysiology of oxidation of LDL.- p53: DNA damage, DNA repair, and apoptosis.- Biosynthesis of nitric oxide: Dependence on pteridine metabolism.- Cytochrome P450: Structure, function, and generation of reactive oxygen species.- DNA damage profiles induced by oxidizing agents.- Depolarization - Transcription coupling in excitable cells.
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