Acute Respiratory Distress Syndrome (eBook, PDF)
Cellular and Molecular Mechanisms and Clinical Management
Redaktion: Matalon, Sadis; Sznajder, Jacob Iasha
40,95 €
40,95 €
inkl. MwSt.
Sofort per Download lieferbar
20 °P sammeln
40,95 €
Als Download kaufen
40,95 €
inkl. MwSt.
Sofort per Download lieferbar
20 °P sammeln
Jetzt verschenken
Alle Infos zum eBook verschenken
40,95 €
inkl. MwSt.
Sofort per Download lieferbar
Alle Infos zum eBook verschenken
20 °P sammeln
Acute Respiratory Distress Syndrome (eBook, PDF)
Cellular and Molecular Mechanisms and Clinical Management
Redaktion: Matalon, Sadis; Sznajder, Jacob Iasha
- Format: PDF
- Merkliste
- Auf die Merkliste
- Bewerten Bewerten
- Teilen
- Produkt teilen
- Produkterinnerung
- Produkterinnerung
Bitte loggen Sie sich zunächst in Ihr Kundenkonto ein oder registrieren Sie sich bei
bücher.de, um das eBook-Abo tolino select nutzen zu können.
Hier können Sie sich einloggen
Hier können Sie sich einloggen
Sie sind bereits eingeloggt. Klicken Sie auf 2. tolino select Abo, um fortzufahren.
Bitte loggen Sie sich zunächst in Ihr Kundenkonto ein oder registrieren Sie sich bei bücher.de, um das eBook-Abo tolino select nutzen zu können.
Proceedings of a NATO ASI held in Corfu, Greece, June 15-25, 1997
- Geräte: PC
- ohne Kopierschutz
- eBook Hilfe
- Größe: 50.48MB
Proceedings of a NATO ASI held in Corfu, Greece, June 15-25, 1997
Dieser Download kann aus rechtlichen Gründen nur mit Rechnungsadresse in A, B, BG, CY, CZ, D, DK, EW, E, FIN, F, GR, HR, H, IRL, I, LT, L, LR, M, NL, PL, P, R, S, SLO, SK ausgeliefert werden.
Produktdetails
- Produktdetails
- Verlag: Springer US
- Seitenzahl: 432
- Erscheinungstermin: 6. Dezember 2012
- Englisch
- ISBN-13: 9781441986344
- Artikelnr.: 43999923
- Verlag: Springer US
- Seitenzahl: 432
- Erscheinungstermin: 6. Dezember 2012
- Englisch
- ISBN-13: 9781441986344
- Artikelnr.: 43999923
- Herstellerkennzeichnung Die Herstellerinformationen sind derzeit nicht verfügbar.
SECTION 1: SODIUM (Na+) TRANSPORT AND FLUID CLEARANCE ACROSS THE NORMAL AND INJURED LUNGS.- Regulation of the Sodium Pump in Hyperoxic Lung Injury.- Lung Edema Clearance during Hyperoxic Lung Injury.- The Role of Lung Lymphatics in Pulmonary Edema Clearance.- Molecular Biology of Lung Na+ Absorption.- Regulation of Alveolar Sodium Transport by Hypoxia.- Vectorial Movement of Sodium in Lung Alveolar Epithelium: Role and Regulation of Na+,K-ATPase.- The Premature Infant's Inability to Clear Fetal Lung Liquid Results in Respiratory Distress Syndrome (RDS).- Role of Matrix Macromolecules in Controlling Lung Fluid Balance: Transition from a Dry Tissue to Edema**.- Role of the Na+-K+-ATPase ?2 Subunit in Lung Liquid Clearance**.- Aveolar Epithelial Fluid Transport under Normal and Pathological Conditions.- SECTION 2: ROLE OF THE PULMONARY SURFACTANT IN THE PATHOGENESIS AND TREATMENT OF ARDS.- Host Defense Capacities of Pulmonary Surfactant.- Alterations of Pulmonary Surfactant in ARDS-Pathogenic Role and Therapeutic Perspectives.- Surfactant Replacement in Patients with ARDS: Results of Clinical Trials.- Potential Role for Pulmonary Surfactant in Lung Transplantation.- Preparation of Surfactant and Lipid Vectors for Delivery of Proteins and Genes to Tissue.- Recombinant SP-C Based Surfactant Can Improve Lung Function in Saline Lavaged Sheep**.- SECTION 3: MECHANISMS AND MODIFICATION OF ACUTE AND CHRONIC LUNG INJURY.- Gene Therapy for the Acute Respiratory Distress Syndrome.- The Use of Recombinant Antioxidants in Neonatal Respiratory Distress Syndrome.- The Early Inflammatory Response in Acute Respiratory Distress Syndrome (ARDS).- The N-Nitroso-N-Methylurethane Induced Acute Lung Injury Animal Model for ARDS: Minireview.- Hyperoxia Induced Fibrosis in ARDS and Bpd:Current Pathology and Cytokine Profiles.- MAP Kinases in Airway Disease: Standing at the Confluence of Basic and Clinical Science.- Oxidant-Regulated Gene Expression in Inflammatory Lung Disease.- Pulmonary Edema of Mixed or Incompletely Understood Pathogenesis.- Biologic Markers of Acute Lung Injury.- Pathways to Oxidative Cell Death.- Divergent Effects of Hypoxia and Oxidants on Mitochondrial Superoxide Dismutase (MnSOD) Gene Expression.- Cellular Sources of Cytokines in Pulmonary Fibrosis.- Strategies to Speed Lung Healing in ARDS.- Pulmonary Microvascular Occlusion in Aggregate Anaphylaxis**.- The Expression of ICAM-1 and P-Selectin Are Increased following Hemorrhage and Resuscitation in Mice**.- Increased Levels of Markers of Protein Oxidation in Bronchoalveolar Lavage Fluid from Patients with ARDS**.- SECTION 4: VENTILATORY STRATEGIES FOR THE MANAGEMENT OF ACUTE RESPIRATORY DISTRESS SYNDROME (ARDS).- The Use of PEEP in the Treatment of ARDS.- Tracheal Gas Insufflation.- Mechanical Ventilation in ARDS: Good or Bad News?.- Ventilator-Induced Lung Injury in Patients with ARDS.- Mechanical Ventilation for ARDS: Should We Reduce Tidal Volume?.- Endotracheal Tube Resistance and Passive Expiratory Time during Mechanical Ventilation in ARDS.- Use of Liquid Ventilation in Animal Models of ARDS.- Inhaled Prostacyclin for the Treatment of ARDS.- Is There Improvement in the Prognosis of ARDS?.- Effect of Ventilation Strategy on Cytokine Expression in an ex Vivo Lung Model**.- Abnormal Tissue Oxygenation and Cardiovascular Changes in Endotoxaemia: Beneficial Effects of Volume Resuscitation**.- SECTION 5: NITRIC OXIDE: FRIEND AND FOE.- Mechanisms of Nitric Oxide Induced Injury to the Alveolar Epithelium.- Macrophage Killing of Mycoplasmas: Involvement of Surfactant Protein A and Nitric Oxide.- Exhaled Nitric Oxide in Patients Undergoing Cardiothoracic Surgery: A New Diagnostic Tool?.- ARDS: Nitric Oxide as Cause and Therapy in Multiple Organ Failure (MOF).- Role of Reactive Nitrogen Species in Asbestos-Induced Pleuro-Pulmonary Injury.- Biomarkers of Oxidative Stress in Adult Respiratory Distress Syndrome.- Reactive Oxygen and Nitrogen Species and Adult Respiratory Distress Syndrome (ARDS): New Mechanisms to Be Considered.- SECTION 6: HEALTH CARE LEGISLATION.- Health and Science Legislation in the United States.
SECTION 1: SODIUM (Na+) TRANSPORT AND FLUID CLEARANCE ACROSS THE NORMAL AND INJURED LUNGS.- Regulation of the Sodium Pump in Hyperoxic Lung Injury.- Lung Edema Clearance during Hyperoxic Lung Injury.- The Role of Lung Lymphatics in Pulmonary Edema Clearance.- Molecular Biology of Lung Na+ Absorption.- Regulation of Alveolar Sodium Transport by Hypoxia.- Vectorial Movement of Sodium in Lung Alveolar Epithelium: Role and Regulation of Na+,K-ATPase.- The Premature Infant's Inability to Clear Fetal Lung Liquid Results in Respiratory Distress Syndrome (RDS).- Role of Matrix Macromolecules in Controlling Lung Fluid Balance: Transition from a Dry Tissue to Edema**.- Role of the Na+-K+-ATPase ?2 Subunit in Lung Liquid Clearance**.- Aveolar Epithelial Fluid Transport under Normal and Pathological Conditions.- SECTION 2: ROLE OF THE PULMONARY SURFACTANT IN THE PATHOGENESIS AND TREATMENT OF ARDS.- Host Defense Capacities of Pulmonary Surfactant.- Alterations of Pulmonary Surfactant in ARDS-Pathogenic Role and Therapeutic Perspectives.- Surfactant Replacement in Patients with ARDS: Results of Clinical Trials.- Potential Role for Pulmonary Surfactant in Lung Transplantation.- Preparation of Surfactant and Lipid Vectors for Delivery of Proteins and Genes to Tissue.- Recombinant SP-C Based Surfactant Can Improve Lung Function in Saline Lavaged Sheep**.- SECTION 3: MECHANISMS AND MODIFICATION OF ACUTE AND CHRONIC LUNG INJURY.- Gene Therapy for the Acute Respiratory Distress Syndrome.- The Use of Recombinant Antioxidants in Neonatal Respiratory Distress Syndrome.- The Early Inflammatory Response in Acute Respiratory Distress Syndrome (ARDS).- The N-Nitroso-N-Methylurethane Induced Acute Lung Injury Animal Model for ARDS: Minireview.- Hyperoxia Induced Fibrosis in ARDS and Bpd:Current Pathology and Cytokine Profiles.- MAP Kinases in Airway Disease: Standing at the Confluence of Basic and Clinical Science.- Oxidant-Regulated Gene Expression in Inflammatory Lung Disease.- Pulmonary Edema of Mixed or Incompletely Understood Pathogenesis.- Biologic Markers of Acute Lung Injury.- Pathways to Oxidative Cell Death.- Divergent Effects of Hypoxia and Oxidants on Mitochondrial Superoxide Dismutase (MnSOD) Gene Expression.- Cellular Sources of Cytokines in Pulmonary Fibrosis.- Strategies to Speed Lung Healing in ARDS.- Pulmonary Microvascular Occlusion in Aggregate Anaphylaxis**.- The Expression of ICAM-1 and P-Selectin Are Increased following Hemorrhage and Resuscitation in Mice**.- Increased Levels of Markers of Protein Oxidation in Bronchoalveolar Lavage Fluid from Patients with ARDS**.- SECTION 4: VENTILATORY STRATEGIES FOR THE MANAGEMENT OF ACUTE RESPIRATORY DISTRESS SYNDROME (ARDS).- The Use of PEEP in the Treatment of ARDS.- Tracheal Gas Insufflation.- Mechanical Ventilation in ARDS: Good or Bad News?.- Ventilator-Induced Lung Injury in Patients with ARDS.- Mechanical Ventilation for ARDS: Should We Reduce Tidal Volume?.- Endotracheal Tube Resistance and Passive Expiratory Time during Mechanical Ventilation in ARDS.- Use of Liquid Ventilation in Animal Models of ARDS.- Inhaled Prostacyclin for the Treatment of ARDS.- Is There Improvement in the Prognosis of ARDS?.- Effect of Ventilation Strategy on Cytokine Expression in an ex Vivo Lung Model**.- Abnormal Tissue Oxygenation and Cardiovascular Changes in Endotoxaemia: Beneficial Effects of Volume Resuscitation**.- SECTION 5: NITRIC OXIDE: FRIEND AND FOE.- Mechanisms of Nitric Oxide Induced Injury to the Alveolar Epithelium.- Macrophage Killing of Mycoplasmas: Involvement of Surfactant Protein A and Nitric Oxide.- Exhaled Nitric Oxide in Patients Undergoing Cardiothoracic Surgery: A New Diagnostic Tool?.- ARDS: Nitric Oxide as Cause and Therapy in Multiple Organ Failure (MOF).- Role of Reactive Nitrogen Species in Asbestos-Induced Pleuro-Pulmonary Injury.- Biomarkers of Oxidative Stress in Adult Respiratory Distress Syndrome.- Reactive Oxygen and Nitrogen Species and Adult Respiratory Distress Syndrome (ARDS): New Mechanisms to Be Considered.- SECTION 6: HEALTH CARE LEGISLATION.- Health and Science Legislation in the United States.