Recently, the CDC issued an alert indicating that autism may impact one out of every150 children. While the syndrome is still officially labeled as idiopathic, research shows the cause of autism may possess genetic, environmental, and possibly immunological factors and that it has at least a correlation to oxidative stress. This book collects work from researchers who report on evidence indicating links between autism and a number of oxidative stress-related abnormalities such as neuroinflammation, mitochondrial dysfunction, membrane and metabolism abnormalities, and signal transduction. The text also reports on potential avenues for new therapeutic intervention strategies.
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