Molecular Mechanisms of Mechanotransduction in Cardiac Cells.- Titin and Titin-Associated Proteins in Myocardial Stress-Sensing and Mechanical Dysfunction.- Mechanical Stretch-Induced Reorganization of the Cytoskeleton and the Small GTPase Rac-1 in Cardiac Fibroblasts.- Molecular Signaling Mechanisms of Myocardial Stretch: Implications for Heart Disease.- Mechanical Stress Induces Cardiomyocyte Hypertrophy Through Agonist-Independent Activation of Angiotensin II Type 1 Receptor.- Mechanically Induced Potentials and Currents of the Cardiac Cells in Healthy and Diseased Myocardium.- Mechanostransduction in Cardiac and Stem-Cell Derived Cardiac Cells.- Stretch-Activated Channels in the Heart: Contribution to Cardiac Performance.- Effects of Applied Stretch on Native and Recombinant Cardiac Na+ Currents.- Mechanosensitive Alterations of Action Potentials and Membrane Currents in Healthy and Diseased Cardiomyocytes: Cardiac Tissue and Isolated Cell.- The Role of Mechanosensitive Fibroblasts in the Heart: Evidence from Acutely Isolated Single Cells, Cultured Cells and from Intracellular Microelectrode Recordings on Multicellular Preparations from Healthy and Diseased Cardiac Tissue.- Scanning Ion Conductance Microscopy for Imaging and Mechanosensitive Activation of Selected Areas of Live Cells.- Mechano-Electric Feedback in the Whole Heart and a Computer Simulation Study.- The Contribution of MEF to Electrical Heterogeneity and Arrhythmogenesis.- Mechanical Modulation of a Reentrant Arrhythmia: The Atrial Flutter Case.- Early Hypertrophic Signals After Myocardial Stretch. Role of Reactive Oxygen Species and the Sodium/Hydrogen Exchanger.- Stretch-Induced Inotropy in Atrial and Ventricular Myocardium.- Effects of Wall Stress on the Dynamics of Ventricular Fibrillation: A Computer Simulation Study of Mechanoelectric Feedback.- Electromechanical Modelling of Cardiac Tissue.- Arteries as a Source of Myogenic Contractile Activity: Ionic Mechanisms.- Specific Mechanotransduction Signaling Involved in Myogenic Responses of the Cerebral Arteries.
Molecular Mechanisms of Mechanotransduction in Cardiac Cells.- Titin and Titin-Associated Proteins in Myocardial Stress-Sensing and Mechanical Dysfunction.- Mechanical Stretch-Induced Reorganization of the Cytoskeleton and the Small GTPase Rac-1 in Cardiac Fibroblasts.- Molecular Signaling Mechanisms of Myocardial Stretch: Implications for Heart Disease.- Mechanical Stress Induces Cardiomyocyte Hypertrophy Through Agonist-Independent Activation of Angiotensin II Type 1 Receptor.- Mechanically Induced Potentials and Currents of the Cardiac Cells in Healthy and Diseased Myocardium.- Mechanostransduction in Cardiac and Stem-Cell Derived Cardiac Cells.- Stretch-Activated Channels in the Heart: Contribution to Cardiac Performance.- Effects of Applied Stretch on Native and Recombinant Cardiac Na+ Currents.- Mechanosensitive Alterations of Action Potentials and Membrane Currents in Healthy and Diseased Cardiomyocytes: Cardiac Tissue and Isolated Cell.- The Role of Mechanosensitive Fibroblasts in the Heart: Evidence from Acutely Isolated Single Cells, Cultured Cells and from Intracellular Microelectrode Recordings on Multicellular Preparations from Healthy and Diseased Cardiac Tissue.- Scanning Ion Conductance Microscopy for Imaging and Mechanosensitive Activation of Selected Areas of Live Cells.- Mechano-Electric Feedback in the Whole Heart and a Computer Simulation Study.- The Contribution of MEF to Electrical Heterogeneity and Arrhythmogenesis.- Mechanical Modulation of a Reentrant Arrhythmia: The Atrial Flutter Case.- Early Hypertrophic Signals After Myocardial Stretch. Role of Reactive Oxygen Species and the Sodium/Hydrogen Exchanger.- Stretch-Induced Inotropy in Atrial and Ventricular Myocardium.- Effects of Wall Stress on the Dynamics of Ventricular Fibrillation: A Computer Simulation Study of Mechanoelectric Feedback.- Electromechanical Modelling of Cardiac Tissue.- Arteries as a Source of Myogenic Contractile Activity: Ionic Mechanisms.- Specific Mechanotransduction Signaling Involved in Myogenic Responses of the Cerebral Arteries.
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