The Molecular Biology of Down Syndrome (eBook, PDF)
Redaktion: Lubec, G.
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The Molecular Biology of Down Syndrome (eBook, PDF)
Redaktion: Lubec, G.
- Format: PDF
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- Größe: 43.47MB
Produktdetails
- Verlag: Springer Vienna
- Seitenzahl: 366
- Erscheinungstermin: 1. Dezember 2013
- Englisch
- ISBN-13: 9783709163801
- Artikelnr.: 53103950
Dieser Download kann aus rechtlichen Gründen nur mit Rechnungsadresse in A, B, BG, CY, CZ, D, DK, EW, E, FIN, F, GR, HR, H, IRL, I, LT, L, LR, M, NL, PL, P, R, S, SLO, SK ausgeliefert werden.
Molecular abnormalities of the brain in Down Syndrome: relevance to Alzheimer's neurodegeneration.- Reduced aldehyde dehydrogenase levels in the brain of patients with Down Syndrome.- The Down Syndrome critical region.- Neuropathology.- c-fos expression in brains of patients with Down Syndrome.- Neuronal cell death in Down's syndrome.- Altered Gene expression in fetal Down Syndrome brain as revealed by the Gene huntinG technique of subtractive hybridization.- Gene expression in fetal Down Syndrome brain as revealed by subtractive hybridization.- Molecular misreadinG of Genes in Down syndrome as a model for the Alzheimer type of neurodegeneration.- Expression of the dihydropyrimidinase related protein 2 (DRP-2) in Down Syndrome and Alzheimer's disease brain is downregulated at the mRNA and dysregulated at the protein level.- Gene expression relevant to Down Syndrome: problems and approaches.- Isolation and analysis of chromosome 21 Genes potentially involved in Down Syndrome.- Differential display reveals deteriorated mRNA levels of NADH3 (complex I) in cerebellum of patients with Down Syndrome.- Serotonin (5-HT) in brains of adult patients with Down Syndrome.- Mechanisms of neuronal death in Down's syndrome.- Impaired brain Glucose metabolism in patients with Down Syndrome.- Oxidative stress and neural dysfunction in Down Syndrome.- Expression of the transcription factor ETS2 in brain of patients with Down Syndrome - evidence against the overexpression-Gene dosage hypothesis.- Neuronal apoptosis inhibitory protein (NAIP)-like immunoreactivity in brains of adult patients with Down Syndrome.- The "Gene dosage effect" hypothesis versus the "amplified developmental instability" hypothesis in Down Syndrome.- Downregulation of the transcription factorscleraxis in brain of patients with Down Syndrome.- Heat-shock protein 70 levels in brain of patients with Down Syndrome and Alzheimer's disease.- Increased levels of 14-3-3 Gamma and epsilon proteins in brain of patients with Alzheimer's disease and Down Syndrome.- Application of Alu-splice PCR on chromosome 21: DSCR1 and Intersectin.- Overexpression of DNAse I in brain of patients with Down Syndrome.
Molecular abnormalities of the brain in Down Syndrome: relevance to Alzheimer's neurodegeneration.- Reduced aldehyde dehydrogenase levels in the brain of patients with Down Syndrome.- The Down Syndrome critical region.- Neuropathology.- c-fos expression in brains of patients with Down Syndrome.- Neuronal cell death in Down's syndrome.- Altered Gene expression in fetal Down Syndrome brain as revealed by the Gene huntinG technique of subtractive hybridization.- Gene expression in fetal Down Syndrome brain as revealed by subtractive hybridization.- Molecular misreadinG of Genes in Down syndrome as a model for the Alzheimer type of neurodegeneration.- Expression of the dihydropyrimidinase related protein 2 (DRP-2) in Down Syndrome and Alzheimer's disease brain is downregulated at the mRNA and dysregulated at the protein level.- Gene expression relevant to Down Syndrome: problems and approaches.- Isolation and analysis of chromosome 21 Genes potentially involved in Down Syndrome.- Differential display reveals deteriorated mRNA levels of NADH3 (complex I) in cerebellum of patients with Down Syndrome.- Serotonin (5-HT) in brains of adult patients with Down Syndrome.- Mechanisms of neuronal death in Down's syndrome.- Impaired brain Glucose metabolism in patients with Down Syndrome.- Oxidative stress and neural dysfunction in Down Syndrome.- Expression of the transcription factor ETS2 in brain of patients with Down Syndrome - evidence against the overexpression-Gene dosage hypothesis.- Neuronal apoptosis inhibitory protein (NAIP)-like immunoreactivity in brains of adult patients with Down Syndrome.- The "Gene dosage effect" hypothesis versus the "amplified developmental instability" hypothesis in Down Syndrome.- Downregulation of the transcription factorscleraxis in brain of patients with Down Syndrome.- Heat-shock protein 70 levels in brain of patients with Down Syndrome and Alzheimer's disease.- Increased levels of 14-3-3 Gamma and epsilon proteins in brain of patients with Alzheimer's disease and Down Syndrome.- Application of Alu-splice PCR on chromosome 21: DSCR1 and Intersectin.- Overexpression of DNAse I in brain of patients with Down Syndrome.