Andere Kunden interessierten sich auch für
![TIME COURSE OF NEURONAL DAMAGE AND RECOVERY INDUCED BY MDMA (ECSTASY)]( "TIME COURSE OF NEURONAL DAMAGE AND RECOVERY INDUCED BY MDMA (ECSTASY)")
TIME COURSE OF NEURONAL DAMAGE AND RECOVERY INDUCED BY MDMA (ECSTASY)38,99 €![Modulation of cell survival by altering mtDNA damage and repair]( "Modulation of cell survival by altering mtDNA damage and repair")
Modulation of cell survival by altering mtDNA damage and repair41,79 €![ACANTHOCEPHALA AND TISSUE DAMAGE IN THE FISH PARASTROMATEUS NIGER]( "ACANTHOCEPHALA AND TISSUE DAMAGE IN THE FISH PARASTROMATEUS NIGER")
ACANTHOCEPHALA AND TISSUE DAMAGE IN THE FISH PARASTROMATEUS NIGER38,99 €![BIOLOGY OF REPRODUCTIVE AGING]( "BIOLOGY OF REPRODUCTIVE AGING")
BIOLOGY OF REPRODUCTIVE AGING51,99 €![Laser-Induced Damage in Optical Materials]( "Laser-Induced Damage in Optical Materials")
Laser-Induced Damage in Optical Materials282,99 €![Identifying Novel DNA Damage Response Genes Using Functional Genomics]( "Identifying Novel DNA Damage Response Genes Using Functional Genomics")
Identifying Novel DNA Damage Response Genes Using Functional Genomics51,99 €![Risks and management of two opposite GI-pathophysiological damage]( "Risks and management of two opposite GI-pathophysiological damage")
Risks and management of two opposite GI-pathophysiological damage33,99 €
Hormonally active compounds (pesticides, herbicides,
bacterial agents, and synthetic hormones) continue to
be released into the environment. These compounds
are widely spread, highly persistent, bioavailable,
and most importantly present at physiologically
relevant concentrations. Many aquatic and
terrestrial populations have exhibited signs of
distress or have been severely impacted because of
exposure to such pollutants. Apoptosis is a
well-conserved hormonally controlled physiological
pathway that is highly sensitive to exposure to
pollutants and xenobiotics. Endocrine active
compounds can cause disturbances in hormonal control
of apoptosis and alter development or maintenance of
reproductive tissue in growing or reproductively
mature individual. These changes can cause reduced
reproductive capacity in the exposed individual. Here
we offer first evidence that petrochemical
contaminants induce apoptosis in highly proliferating
reproductive, hematopoietic, and immune (thymus, bone
marrow, and spleen) tissue of small mammals living on
contaminated sites; we also show that aquatic
pollution effects on reproductive fitness can be
monitored using in vitro cultures of fish eggs.
bacterial agents, and synthetic hormones) continue to
be released into the environment. These compounds
are widely spread, highly persistent, bioavailable,
and most importantly present at physiologically
relevant concentrations. Many aquatic and
terrestrial populations have exhibited signs of
distress or have been severely impacted because of
exposure to such pollutants. Apoptosis is a
well-conserved hormonally controlled physiological
pathway that is highly sensitive to exposure to
pollutants and xenobiotics. Endocrine active
compounds can cause disturbances in hormonal control
of apoptosis and alter development or maintenance of
reproductive tissue in growing or reproductively
mature individual. These changes can cause reduced
reproductive capacity in the exposed individual. Here
we offer first evidence that petrochemical
contaminants induce apoptosis in highly proliferating
reproductive, hematopoietic, and immune (thymus, bone
marrow, and spleen) tissue of small mammals living on
contaminated sites; we also show that aquatic
pollution effects on reproductive fitness can be
monitored using in vitro cultures of fish eggs.